Lezo is used to treat certain stomach and esophagus problems (such as acid reflux, ulcers). It works by decreasing the amount of acid your stomach makes. It relieves symptoms such as heartburn, difficulty swallowing, and persistent cough. This medication helps heal acid damage to the stomach and esophagus, helps prevent ulcers, and may help prevent cancer of the esophagus. Lezo belongs to a class of drugs known as proton pump inhibitors (PPIs).
Other names for this medication:
Lansoprazole,
Agopton,
Amarin,
Anzo,
Aprazol,
Bamalite,
Compraz,
Degastrol,
Epicur,
Frilans,
Gastrazol,
Gastrex,
Gastride,
Gastrolan,
Helicol,
Ilsatec,
Keval,
Lancap,
Lancid,
Lanfast,
Lanpro,
Lanproton,
Lansacid,
Lansazol,
Lansec,
Lanser,
Lanso,
Lansobene,
Lansohexal,
Lansol,
Lansoloc,
Lansomid,
Lansopral,
Lansoprazol,
Lansoprol,
Lansoptol,
Lansor,
Lansox,
Lanton,
Lanzap,
Lanzol,
Lanzopral,
Lanzoprazol,
Lanzor,
Lanzostad,
Lanzul,
Lapol,
Lapraz,
Laprazol,
Larona,
Lasgan,
Lasoprol,
Lazol,
Leedom,
Lexid,
Lezo,
Limpidex,
Monolitum,
Ogasto,
Ogastoro,
Ogastro,
Opiren,
Prazotec,
Prevacid,
Propump,
Prosogan,
Protica,
Protoner,
Refluxon,
Refluyet,
Solans,
Solox,
Takepron,
Tersen,
Trogas,
Ulceran,
Ulpax,
Zolt,
Zomel,
Zoprol,
Zoton,
Zotrole,
Similar Products:
Topcef,
Aplenzin,
Teraprost,
Diflazon,
Pioglitazone,
Sanprima,
Ortofena,
Parkisan,
Betaren,
Klomeprax,
Meprim,
Reslin,
Flegyl,
Ledar,
Exomax,
Degastrol,
Soza,
Ceftin,
Coversum,
Mavicam,
Fokusin,
Valosine,
Lori,
Noell,
Aclonac,
Kaptin,
Ketokonazol,
Aciphex,
Metoprolol,
Klindacin,
Isox,
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The genomes (188 kbp) of the prototype Molluscum contagiosum virus type 1 (MCV-1) and a variant strain (MCV-1v) were characterized by construction of the physical maps of the viral DNA for the restriction enzymes BamHI, ClaI, EcoRI, and HindIII using a defined gene library harboring the DNA sequences of the MCV-1 genome and by DNA-DNA acimed 20 mg prospecto hybridizations. The different photoproducts were formed by a primary photochemical mechanism which involves an initial cleavage of the C-C bond alpha to the carbonyl group, followed by several secondary processes. However, the insertion of a 15-min delay in stimulation resulted in a reduction in the intensity of the sensations produced by further stimulation, i.e., desensitization occurred. In addition, we also found that excessive expression of PDI and/or BiP could decrease the mRNA expression of pglyrp-1 gene.