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Ligand-Specific Transcriptional Mechanisms Underlie Aryl Hydrocarbon Receptor-Mediated Developmental Toxicity of Oxygenated PAHs. These functional changes appear to contribute to the hyperalgesia and spontaneous pain associated with tissue injury. Induction of IL-1: independent production of IL-1 alpha and IL-1 beta. The RAOV group showed a lower CSA of gastrocnemius muscle fibers compared to RA and CT-Sham groups.
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